Written in English
|The Physical Object|
|Pagination||viii, 43 l.|
|Number of Pages||43|
Kindig D, Spargo B, Kirsten WH. Glomerular response in Aleutian disease of mink. Lab Invest. Mar; 16 (3)– Henson JB, Gorham JR, Padgett GA, Davis WC. Pathogenesis of the glomerular lesions in aleutian disease of mink. Immunofluorescent studies. Arch Pathol. Jan; 87 (1)–Cited by: Parallel studies of glomerular lesions in Aleutian disease (AD) in both Aleutian and non-Aleutian genotypes of mink by histopathological and immunofluorescence techniques revealed three kinds of lesions: mild, proliferative and by: Abstract. The events occurring in glomerulonephritis produce a complex and complicated pattern of glomerular changes. Though these changes can be readily appreciated by light microscopy, many are too subtle to be clearly by: Karstad, L.: Viral plasmacytosis (Aleutian disease) in mink. V. The occurrence of hyalin glomerular lesions and fibrinoid arteritis in experimental infections. Canad. J. comp. Med. 29, 66–74 (). PubMed Google ScholarCited by:
Henson JB, Gorham JR, Padgett GA, Davis WC. Pathogenesis of the glomerular lesions in aleutian disease of mink. Immunofluorescent studies. Arch Pathol. Jan; 87 (1)– McGuire TC, Crawford TB, Henson JB, Gorham JR. Aleutian disease of mink: detection of large quantities of complement-fixing antibody to viral antigen. J by: The glomerular tip lesion is a segmental histological glomerular abnormality that occurs in some patients with proteinuria and usually nephrotic syndrome.1, 2, 3 First described by Howie and Brewer 1 in , but observed on renal biopsy specimens dating back to , 3 the tip lesion is initially manifest as prominent vacuolated podocytes and intracapillary foam cells at or adjacent to the Cited by: Morel-Maroger L () Glomerular lesions induced by interferon. Transplant Proc – PubMed Google Scholar Morel-Maroger L, Sloper JC, Vinter MI, Biol MI, Woodrow D, Gresser I () An ultrastructural study of the development of nephritis in mice treated with interferon in . Patterns of glomerular lesions. Glomerular changes can be focal (only in some glomeruli) or diffuse (in all or almost all the glomeruli), and segmental (only a part of the glomerulus) or global (the entire glomerulus). The first step in the evaluation of the glomerular alterations is to .
In this study, repeated vaccination of mink with live-virus va ccines was investigated as a model for vaccine-induced glomerular injury. Three groups of wk-old mink, 15 per group, were. This content is provided as a service of the National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK), part of the National Institutes of Health. The NIDDK translates and disseminates research findings to increase knowledge and understanding about health and disease among patients, health professionals, and the public. Overview of the Current Approach to Glomerular Disease Classification. In book: Glomerulonephritis, pp A large study showed E lesions are predictive of outcome in children and adults. Studies are now in progress to determine how the individual histo- logical features correlate with the clinical and labora- tory data. It is already obvious that the focal sclerosing glomerular lesions and various forms of proliferative glomerulonephritis account for the majority of steroid non-responders and cases of renal by: